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Tuesday 6 June 2017

Atheistic science's love/hate relationship with the design inference.

Fairy Circles? The Design Inference at Work
Evolution News & Views


From time to time we point out illustrations of how scientists outside the ID movement use the design inference, demonstrating how natural and pervasive design reasoning is. Everyone, scientist or layman, uses such reasoning. It's both valid, when correctly applied, and inescapable.



In Undeniable , Douglas Axe talks about the "common science" that even children engage in, employing what he calls a "universal design intuition" with which we are all born. Axe is careful not to take this too far, but he argues that it's the default for human beings, even as materialists try to explain it away with question-begging Darwinian just-so stories.



In past articles, we've looked at scientists trying to understand the origin of rock piles under the Sea of Galilee or on a mountainside. We've talked about SETI. . And we puzzled over curious lines of rocks in the Jordanian desert. We too have been careful not to jump to conclusions, as two examples failed the design filter, and perfectly natural things like sandstone arches can be quite symmetrical and artistic, but still explainable by natural law and chance. The examples show that one can infer design without knowing anything about the designer.



Today's case study is a doozy. It's most likely a natural phenomenon, but to this day, scientists have been unable to explain it. Some are not ruling out intelligent design. Megan Gannon at Live Science introduces the phenomenon:



"Fairy circles" are regular, repeating patches of dirt in remote grasslands that, when viewed from above, look like whimsical rings that were scattered across a landscape. Despite their fanciful appearance, the patterns have been a source of serious scientific debate for the last four decades. While some have argued that the geometric patterns are the work of termites, others have postulated that the circles form naturally as vegetation self-organizes in competition for scarce water and other nutrients. [Emphasis added.]

Adding to the confusion is the fact that these orderly rings have been found in Namibia and in the Australian outback, but may or may not have the same cause. Here's a sampling of some recent literature on the subject. Notice the first article's premature celebration.


"Mystery of Desert 'Fairy Circles' Solved, Creators Found" (Live Science)



"Are Namibian 'Fairy Circles' the Consequence of Self-Organizing Spatial Vegetation Patterning?" (PLOS ONE)



"Mysterious 'Fairy Circles' Not Explained by Termites, Study Suggests" (Live Science)



"Gradual regime shifts in fairy circles" (PNAS)



"Experiments Testing the Causes of Namibian Fairy Circles" (PLOS ONE, 10/28/15)



"Discovery of fairy circles in Australia supports self-organization theory" (PNAS)



"Are 'Fairy Circles' Just the 'Ghosts' of Termite Nests?" (Live Science)



"Fairy circles or ghosts of termitaria? Pavement termites as alternative causes of circular patterns in vegetation of desert Australia" (PNAS)



It's hard not to chuckle at the talk of fairies in the august Proceedings of the National Academy of Sciences, knowing as we do that Richard Dawkins has often ridiculed the religious as believing in "fairies, goblins, hobgoblins, leprechauns" and other mythical creatures. But obviously it's just a figure of speech here.



Unlike the crop-circle craze, which many suspected from the beginning involved human design (later proved by eyewitnesses and the increasing elaboration of the patterns), this one is not so obvious. The default explanation has been to infer something natural. But what? Until a natural cause is demonstrated, scientists were not able to rule out an intelligent cause.



The last paper, from PNAS, appears to have nailed the solution, at least for the circles in Australia. They are the remains of active or abandoned nests from underground termites. It took years of careful observation to arrive at that conclusion, however, passing through theories of self-organization, tribal artwork or farming patterns, hydrocarbon seeps, or products of carnivorous ants.


It's still not clear that termites explain the Namibian circles. Even now, not everyone is convinced about the termite theory. The point is that the problem has occupied the attention of scientists from around the world, rightly so, who have published in leading journals. And so design science marches on. No apologies needed.

Epigenetics v. the walking dead.

Nematodes Push Epigenetics to the Limit, as Another “Zombie” Bites the Dust
David Klinghoffer | @d_klinghoffer

Evolution as “settled science” in the mind of the public and the media is based a range of persistent myths – documented by  Jonathan Wells  in his new book  Zombie Science: More Icons of Evolution, the sequel to Icons of Evolution. One myth, a shambling zombie if ever there were one, is what Dr. Wells calls in Chapter 4, “DNA – The Secret of Life.”

But isn’t DNA the one and only secret of life? Neo-Darwinism, a gene-obsessed doctrine, needs it to be. But increasing aware of epigenetic — that is, bypassing genetic – inheritance messes with the simple story that evolutionists have successfully sold up till now. New research with roundworms pushes the extent of epigenetic effects farther than they’ve ever been observed before – 14 generations.

By switching the worms back and forth between cooler and warmer environments, European scientists were able to induce an inherited change in gene expression producing a fluorescent glow. Nematodes, especially the species C. elegans, are a favorite with researchers because of their brief lives and the consequent rapid passing of generations. A phenomenon of comparable extent in humans would be much harder to document.

The work was reported in Science. They call it “environmental information” (“Transgenerational transmission of environmental information in Caenorhabditis elegans”):

Ancestral legacy effects

Environmental change can critically affect the lifestyle, reproductive success, and life span of adult animals and their for generations. Klosin et al. showed that in the nematode worm Caenorhabditis elegans, exposure to high temperatures led to expression of endogenously repressed copies of genes — sometimes called “junk” DNA. This effect persisted for >10 generations of worms. The changes in chromatin occurred in the early embryo before the onset of transcription and were inherited through eggs and sperm.
Note the reference to “Junk DNA,” another zombie of evolutionary science dealt with by Jonathan Wells another book, The Myth of Junk DNA.

From the story at  Science Alert, lucidly explained by Signe Dean:

To study how long the environment can leave a mark on genetic expression, a team led by scientists from the European Molecular Biology Organisation (EMBO) in Spain took genetically engineered nematode worms that carry a  transgene for a fluorescent protein. When activated, this gene made the worms glow under ultraviolet light.

Then, they switched things up for the nematodes by changing the temperature of their containers. When the team kept nematodes at 20° Celsius (68° F), they measured low activity of the transgene — which meant the worms hardly glowed at all.

But by moving the worms to a warmer climate of 25° C (77° F), they suddenly lit up like little wormy Christmas trees, which meant the fluorescence gene had become much more active.

Their tropical vacation didn’t last long, however. The worms were moved back to cooler temperatures to see what would happen to the activity of the fluorescence gene.

Surprisingly, they continued to glow brightly, suggesting they were retaining an ‘environmental memory’ of the warmer climate — and that the transgene was still highly active.

Furthermore, that memory was passed onto their offspring for seven brightly-glowing generations, none of whom had experienced the warmer temperatures. The baby worms inherited this  epigenetic change through both eggs and sperm.

The team pushed the results even further — when they kept five generations of nematodes at 25° C (77° F) and then banished their offspring to colder temperatures, the worms continued to have higher transgene activity for an unprecedented 14 generations.
What’s the purpose for the worms? One of researchers, lead author Adam Klosin, says it could be “a form of biological forward-planning.” That doesn’t sound very Darwinian.


Jonathan Wells concludes, “DNA does not contain the genetic program for an organism, and DNA is far from being the secret of life. Continued faith in it is rooted in materialism.” That is, a materialist understanding of evolution requires the dogma of DNA. But science, meanwhile, marches on and another zombie bites the dust.

Yet more on Darwinism's losing on every sale.

Macroevolution by Loss: Flightless Birds Grounded by “Relaxed Selection”
Evolution News @DiscoveryCSC

Evolutionists wanting to make a big deal out of flightlessness in birds are like the merchant who lost money on every sale but tried to make it up in volume.

Watch any documentary about Darwin, and you’re certain to get scenes of animals on the Galápagos Islands. In the panorama of unique creatures, you’re likely to see the flightless cormorant with its pitifully shrunken wings. Darwin made up a story about how this might have been good for the birds, but surely they’d rather fly like all other cormorants. Now for the first time, researchers have now tried to determine what happened genetically…and here’s the reason: they’re sick with a genetic disease.

With the help of a Chilean local expert, eight American scientists looked into the genome of the flightless cormorant, Phalacrocorax harrisi, and compared it with its flying relatives. It’s surprising that since Darwin’s time nobody had a clue what caused the change in this icon of evolution. The Introduction to the paper in  Science Magazine states:

Changes in the size and proportion of limbs and other structures have played a key role in the evolution of species. One common class of limb modification is recurrent wing reduction and loss of flight in birds. Indeed, Darwin used the occurrence of flightless birds as an argument in favor of his theory of natural selection. Loss of flight has evolved repeatedly and is found among 26 families of birds in 17 different orders. Despite the frequency of these modifications, we have a limited understanding of their underpinnings at the genetic and molecular levels. [Emphasis added.]
Well, it’s about time to check Darwin on his speculative idea. Phys.org introduces the hero:

The flightless cormorant is one of a diverse array of animals that live on the Galapagos Islands, which piqued Charles Darwin’s scientific curiosity in the 1830s. He hypothesized that altered evolutionary pressures may have contributed to the loss of the ability to fly in birds like the Galapagos cormorant.
We see the Darwinian escape valve at work again. It’s crystalized in those two words, “may have.” Anything may have happened. Pigs may have flown millions of years ago, when nobody could observe them. As luck would have it, they didn’t leave any fossils. But altered evolutionary selection pressures “may have” contributed to pigs’ loss of ability to fly.

To Darwinians, “evolutionary pressure” can point in any direction: up, down, or sideways. But think again. Some forces, like gravity, point only down. Suppose we suggested that gravity can point in any direction: sometimes making pigs fly. That would be ridiculous. Darwin needs to prove that selection pressure can point up toward innovation and progress; he can’t merely assume it. But we’ll take him up on this example of negative selection. Sure, evolution can break things, and it did so in the case of flightless cormorants.

Phys.org explains what broke:

The team also found that the flightless cormorants have an abnormally high number of genetic mutations affecting cilia — small, hair-like structures that protrude from cells and regulate everything from normal development to reproduction.

Cilia play a critical role in bone growth. People born with skeletal ciliopathies have shorter limbs, narrowed chests and stunted rib cages — as do the Galapagos cormorants. The UCLA results suggest that CUX1 controls many aspects of cilia, some of which influence bone growth.
These birds are sick. They continue to breed because the genetic sickness has spread to the whole colony, and there’s nobody else to breed with. They have what’s called ciliopathy, or a genetic disease that hinders the healthy development of cilia. Cilia, remember, are examples of irreducible complexity that Michael Behe described in his book, Darwin’s Black Box. Almost all cells have these molecular machines; some are motile, like those that sweep dust out of the airways, but many are stationary, acting like antennae. A lack of healthy cilia, when not fatal, leads to numerous genetic diseases. One feels sorry for these poor birds, but good icons of evolution they are not.

Some evolutionists try to rescue Darwin in this case by suggesting that the genetic disease helps the birds in some way: “changes that lead to flightlessness may help birds survive because they enhance their ability to do something else, like swimming — so-called positive selection.” Nice try, but more likely, selection pressure for costly flight and wings was “relaxed” because of a lack of predators on the island. “When flying isn’t essential for survival, the mutations that hinder flight can gradually accumulate in the gene pool.” Lives there a Darwinist who would consider this a helpful argument for universal common descent by mutation and selection?

“Relaxed selection” is like relaxed maintenance on your car. Without upkeep, it will rust and parts will break. You still may be able to drive it a little, but relaxation is not going to build a better car, or build a car in the first place.

The researchers found a way to manipulate the genetic data to affirm that 3 out of 11 genes showed evidence of “positive selection toward flightlessness.” They do this not by measuring actual benefits to the bird, but just measuring the ratio of synonymous to non-synonymous mutations. “These results suggest that selection toward flightlessness may be partially responsible for the phenotype of P. harrisi.” The results “suggest” that it “may” be “partially” responsible. Let’s try that on our relaxed-maintenance automobile: “The results suggest that positive selection toward rust may have been responsible for the car’s inability to drive faster than 20 mph.” Hooray! Evolution confirmed!

In a last-ditch effort to rescue Darwin, the research team suggested that the birds are stuck in a Peter-Pan-like juvenile stage, a condition called “heterochrony” (“other-timing”). Scientific jargon loses some of its power to impress when you translate it out of Latin. They also dazzle with “paedomorphosis” — “baby shape” (pedo as in pediatrician, morph for shape or form). Translation: “other-timing” led to “baby-shape.” Question: What, exactly, does that explain, other than to conceal the observation in a language most people don’t understand? And what does it have to do with Darwinian evolution?

Although we have identified multiple variants that likely contribute to the flightless phenotype of P. harrisi, we cannot exclude the possibility that other genes and pathways may contribute to the phenotype, nor the contribution of noncoding regulatory variants. Further characterization of the individual and joint contributions of the variants found in this study will help us to reconstruct the chain of events leading to flightlessness and to genetically dissect macroevolutionary change. We hypothesize that mutations in cilia or functionally related genes could be responsible for limb and other skeletal heterochronic transformations in birds and diverse organisms, including humans.
All is not in vain. At least we now know a little bit about genetic variants in flightless cormorants compared to flying cormorants. That’s interesting. We’ve learned part of what broke to make the birds genetically sick and grounded. But if there was any macroevolution happening, it was downwards, from the skies to the dirt, from working cilia to broken cilia. If there was a transformation, it was from powered flight to pitiful flopping.


It’s a loss every way you look at it. But don’t worry; Darwin will make it up in volume!